Complication | Evidence | PMID |
Nephropathy | 1. eNOS and CD34 expression was upregulated in H. pylori-positive controls but not in H. pylori-positive diabetic patients (p=0.010 and 0.007 for the corpus and p=0.036 and 0.047 for the antrum, respectively). | 15737001 |
Cardiovascular | 1. Our provide evidence that up-regulation of CD36 may have a role in increasing oxidative stress in microvascular endothelial cells (MVECs) and the heart in chronic diabetes. | 15915335 |
Atherosclerosis | 1. Incubation of monocytes with AGE + bovine serum albumin (BSA), CD36 expression and intracellular ROS increased significantly in all groupsAdvanced glycation end products (AGEs) engagement of a monocyte surface receptor (RAGE) induces atherosclerosis. AGEs also act as CD36 ligands.Blocking RAGE expression using siRNA in human monocytes led to a significant inhibition of CD36 expression and ROS production, suggesting a positive interaction between RAGE, CD36 expression and ROS generation in monocytes. | 19190027 |
Insulin resistance and Inflammation | 1. Mice harboring CD36-specific deletion in hematopoietic-derived cells (HSC CD36KO) fed an HFD displayed improved insulin signaling and reduced macrophage infiltration in adipose tissue compared with wild-type mice, but this did not translate into protection against HFD-induced whole-body insulin resistance. | 21378177 |