Go to Advanced Search
MOK |
LOCUS ID | 5891 | |||||||||||||||||||
GENE_SYMBOL | MOK | |||||||||||||||||||
GENE NAME | MOK protein kinase | |||||||||||||||||||
SYNONYMNS | RAGE, RAGE1, STK30, RAGE-1 | |||||||||||||||||||
CHROMOSOME | 14 | |||||||||||||||||||
HOMOLOGENE ID | 8062 |
microRNAs | NA | NA |
GENE SUMMARY |
---|
This gene belongs to the MAP kinase superfamily. The gene was found to be regulated by caudal type transcription factor 2 (Cdx2) protein. The encoded protein, which is localized to epithelial cells in the intestil crypt, may play a role in growth arrest and differentiation of cells of upper crypt and lower villus regions. Multiple altertively spliced transcript variants encoding different isoforms have been observed for this gene. [provided by RefSeq, Dec 2012] |
OBSERVATIONS |
---|
Complication | Evidence | PMID |
Atherosclerosis | 1. Advanced glycation end products (AGEs) and their receptor (RAGE) play an important role in accelerated atherosclerosis in diabetes.Serum levels of sRAGE are positively associated with MCP-1 and TNF-alpha levels in type 2 diabetic patients. These observations suggest the possibility that sRAGE level may become a novel biomarker of vascular inflammation in type 2 diabetic patients. | 17592553 |
Nephropathy | 1. The RAGE(2) haplotype is associated with DN in type 2 diabetics and with earlier DN onset and, thus, can be regarded a marker for DN. | 15790669 |
Dyslipidemia | 1. The up-regulation of RAGE was related to dyslipidemia and diabetic nephropathy. | 25324883 |
Insulin resistance and Inflammation | 1. AGE-RAGE interaction contributes to fat accumulation in the liver leading to inflammation, fibrosis, insulin resistance, and other complications of the fatty liver disease.? | 31641351 |