Complication | Evidence | PMID |
Nephropathy | 1. In vivo, intrarel delivery of adenovirus expressing SOCS1 and SOCS3 to diabetic rats significantly improved renal function and reduced renal lesions associated with diabetes, such as mesangial expansion, fibrosis, and influx of macrophages. Suppression of the JAK/STAT pathway by increasing intracellular SOCS proteins may have therapeutic potential in diabetic nephropathy | 20185635 |
Tuberculosis | 1. We studied gene expression in peripheral blood cells of patients with diabetes (n = 10) and healthy endemic controls (EC, n = 11) both with and without MTB infection. Mycobacterial antigen (PPD) and mitogen-stimulated SOCS1, SOCS3, interferon-gamma (IFN-?), IL-6, and tumor necrosis factor alpha (TNF?) mRNA expression levels were determined using real-time polymerase chain reaction. | 28043584 |
Insulin resistance and inflammation | 1. SOCS1 LysM-Cre mice (SOCS1 floxed mice with mice expressing Cre recombinase under the control of the LysM-Cre promoter) had increased macrophage expression of CD11c, enhanced sensitivity to LPS, and palmitic acid and increased serum concentrations of tumor necrosis factor-?, interleukin-6, and monocyte chemoattractant protein. | 21646388 |